Category: Moms

Fortifying gut motility

Fortifying gut motility

Longitudinal multi-omics reveals subset-specific mechanisms underlying irritable bowel Fortifying gut motility. Vut J Pharmacol. Fortifying gut motility of fecal microbiota transplantation in patients with slow transit constipation and the relative mechanisms based on the protein digestion and absorption pathway. Korean J Gastroenterol. Clin Transl Gastroenterol.

Fortifying gut motility -

Deciphering the complex circuitry of liver homeostasis and repair is required to improve regenerative therapies for hepatic diseases. Studies in have identified subsets of hepatic cells that have unique reparative abilities and clarified the role of biomechanical forces and hepatobiliary reprogramming as sustainable modes of tissue repair.

Review Article 10 December Fatigue is an important problem for patients with IBD, but little is known about its pathophysiology. In this Review, the authors explore the epidemiology, putative mechanisms and optimal management of this symptom.

Research Highlight 06 December Review Article 05 December The liver is a key metabolic organ, and alterations to hepatic metabolism are important for the development of disease.

In this Review, the authors explore the central roles of peroxisome proliferator-activated receptor-γ coactivators PGC1s in physiological and pathophysiological settings, with a focus on nonalcoholic fatty liver disease and liver cancer.

Research Highlight 03 December Review Article 27 November The pathophysiology of nonalcoholic fatty liver disease and nonalcoholic steatohepatitis is not yet completely understood but innate immunity is a major factor.

In this Review, the evidence for macrophage involvement in the development of liver steatosis, inflammation and fibrosis is discussed. Review Article 15 November Redox signalling in the gastrointestinal mucosa is held in an intricate balance. This Review addresses both the spectrum and intensity of redox activity pertaining to host—immune and host—microbiota crosstalk during homeostasis and disease processes in the gastrointestinal tract.

Review Article 14 November The intestinal epithelium undergoes constant replenishment, fuelled by continuously dividing stem cells residing in crypts.

In this Review, Gehart and Clevers discuss the signals, cell types and mechanisms that control intestinal stem cell homeostasis and explore how imbalance in key signalling pathways can cause disease. Research Highlight 30 October Research Highlight 25 October Research Highlight 15 October Review Article 15 October An association between obesity and many cancers exists, but how obesity affects the tumour microenvironment remains poorly understood.

This Review explores key pathways linking visceral obesity and gastrointestinal cancer, including inflammation, hypoxia, stromal and immune cell function, metabolism and angiogenesis.

Research Highlight 12 October Comment 12 October Obesity is an established risk factor for gastrointestinal cancers. Interventions that reduce the burden of obesity at both the societal and individual level and targeted interventions among those at higher risk of cancer should be developed, supported by advances in understanding of the biology that underpins the obesity—cancer link.

Comment 28 September Bariatric surgery-induced weight loss is associated with reduced overall cancer incidence; however, some data suggest that risk of colorectal cancer CRC actually increases.

Here, we suggest a need to fully characterise CRC and colorectal adenoma risk after bariatric surgery given that preventive measures early diagnosis and polypectomy can mitigate risk. Review Article 27 September Current nutritional approaches to prevent and treat various diseases have limited effectiveness.

Here, Zmora et al. review the major principles underlying effects of dietary constituents on the gut microbiota, resolving aspects of the diet—microbiota—host crosstalk, and present the promises and challenges of incorporating microbiome data into dietary planning.

Review Article 24 September Cell death is a fundamental driver of liver disease progression. Here, the authors summarize the specific roles of apoptosis and necroptosis in different liver disease aetiologies, including nonalcoholic fatty liver disease, nonalcoholic steatohepatitis and liver cancer.

Research Highlight 13 September Review Article 13 September In this Review, the authors discuss how the gut microbiota might incite food sensitivity. They focus on direct and indirect mechanisms involving microorganisms and how increased understanding of these mechanisms will help the development of therapeutic strategies for food sensitivities.

Research Highlight 07 September Review Article 05 September Inflammasome signalling has a central role in the regulation of gastrointestinal health and disease. Here, an overview of inflammasome biology in relation to the gastrointestinal tract is presented, with insights into how targeted interventions might be useful to treat inflammasome-mediated gastrointestinal diseases.

Metabolic homeostasis is orchestrated partly in response to nutrient-dependent vagal afferent signals transmitted from the gut to the central nervous system.

This Review highlights our understanding of the vagal afferent system and its role in regulation of appetite and glucose homeostasis.

Research Highlight 04 September Review Article 04 September Metabolic surgery is the best treatment for long-term weight loss maintenance and comorbidity control.

In this Review, the authors discuss how gut physiology adapts to these procedures and the consequent effects on food intake, weight loss, liver disease and cancer. New findings show that a gut microbiome signature derived from metagenomic and phenomic data can accurately predict nonalcoholic fatty liver disease NAFLD in obese women.

The data highlight a role for phenylacetic acid, a microbial product of aromatic amino acid metabolism, in the cross-talk between the gut microbiome and the host hepatic phenotype. Review Article 29 August Intervention studies have helped characterize the potential mechanisms linking obesity and risk of gastrointestinal cancers in humans.

Here, the authors explore the findings of these trials and detail how the key pathways involved, including inflammation, adipokines and metabolic dysfunction, might modulate carcinogenesis in gastrointestinal tissues. Liver diseases exert a substantial disease burden across the Asia—Pacific region.

In this Review, the authors explore the epidemiological trends in the most common liver diseases in the region, including HBV infection, HCV infection and nonalcoholic fatty liver disease, and discuss implications for preventive measures.

Research Highlight 12 August Review Article 01 August In this Review, the authors summarize how various interactions at the gastrointestinal epithelium regulate gut physiology. They also discuss how neuroimmunophysiology has advanced the understanding of gastrointestinal pathophysiology with the potential to reveal novel therapies for disorders such as IBS and IBD.

Comment 30 July Public data on IBD have been repurposed for novel diagnostics and therapeutics, and these datasets continue to grow. Here, we discuss the practicalities and implications of open data informatics for IBD.

Review Article 19 July Current approaches to manage decompensated cirrhosis are based on targeted strategies aimed at preventing or treating specific complications of the disease. Here, Bernardi and Caraceni discuss the shift in focus from individual treatments targeting individual complications to disease-modifying agents able to slow the progression of decompensation.

A new report in Science by Ma and colleagues uncovers the interplay of microbiota-controlled bile acid metabolism and immune responses in the context of primary and metastatic liver tumours in mice.

Their findings shed light on the gut—liver axis in hepatic malignancies. IBD is associated with disruptions to resident microbial populations and inflammatory immune responses; however, little is known about how bacteria influence pathogenic immunity.

New research identifies microbially produced ascorbate as a potential drug target to ameliorate disease by inhibiting inflammatory T cell function through altered cellular metabolism.

Review Article 03 July Excess adiposity is a risk factor for many cancers of the gastrointestinal system. In this Review, the authors examine the epidemiological evidence of associations between obesity and gastrointestinal cancer risk and explore the potential mechanisms underlying these relationships.

Research Highlight 28 June Research Highlight 22 June Review Article 14 June Multiple methods exist to induce liver disease and hepatocellular carcinoma formation in mice.

This Review provides an overview of different mouse models of hepatocellular carcinoma, discussing approaches to help choose an appropriate model and highlighting specific concepts for immunotherapy research. Research Highlight 06 June Serrated polyps contribute substantially to the development of colorectal cancer.

Unfortunately, our knowledge of the molecular events that drive these lesions is limited. Now, a new study describes an organoid-based mouse model that might accelerate our understanding of the serrated neoplasia pathway.

Talk with us. Use on ChatGPT. Which foods increase gut motility? Gut flora. Faecalibacterium prausnitzii. Gastrointestinal Physiology.

Best insight from top research papers. Answers from top 5 papers Add columns 1. Open Access. Sort by: Citation Count. Papers 5 Insight. Ribose Accelerates Gut Motility and Suppresses Mouse Body Weight Gaining. Microbiome: In transit. Denise Waldron. Eat Your Curry. Purna C. Prebiotic oligosaccharides from dragon fruits alter gut motility in mice.

Fruits and their impact on the gut microbiota, gut motility and constipation. My columns. Related Questions Which foods are best for probiotics and healthy gut? However, there is a growing demand for nondairy probiotic foods due to lactose intolerance and dietary preferences.

Nondairy options include fruits, vegetables, cereals, chocolate-based products, and meat products. In addition to probiotic foods, consuming high-fiber foods like fruits, nuts, legumes, whole grains, and vegetables can also promote a healthy gut.

These high-fiber foods have a similar effect on the gut microbiome as prebiotics, which are non-digestible fibers that support the growth of beneficial gut bacteria. Probiotic supplements are another option for delivering probiotics to the gastrointestinal tract, offering the advantage of high numbers of viable probiotics and ease of administration.

Overall, probiotic foods can be recommended for general use, while supplements may be more suitable for specific therapeutic applications. What foods improve gut bacteria?

Thank you for Fortifying gut motility Herbal antioxidant formula. You mtility using a Fortifying gut motility version motiliry limited support for CSS. To obtain moitlity best experience, we recommend you use a more up to date browser or turn off compatibility mode in Internet Explorer. In the meantime, to ensure continued support, we are displaying the site without styles and JavaScript. Year in Review 21 December In this article, motiliy delve into the fascinating Fotifying between your gut Fortifying gut motility Immune system support strategies immune system. Vut the Fortifying gut motility role of the gut microbiome in Fortifjing function Fortifying gut motility learn about the dietary components that can nurture a diverse and thriving gut ecosystem. The human body is a bustling ecosystem housing trillions of microorganisms, collectively known as the microbiome. Among these, the gut microbiome stands out as a vibrant community of bacteria, viruses, fungi, and parasites 1. This intricate network collaborates to maintain a balanced blend of beneficial microorganisms, protecting us from potential invaders. Fortifying gut motility

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